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Clinical data
Pronunciation/ f ə ˈ m ɒ t ɪ d iː n /
Trade namesPepcid, others
License dataUS   FDA: Famotidine
Pregnancy categoryAU: B1 US: B (No risk in non-human studies)
Routes of administrationOral (tablets), Intravenous
ATC codeA02BA03 ( WHO )
Legal status
Legal statusAU: S3 (Pharmacist only) /S4; pharmacist only, UK: General sales list (GSL, OTC) US: OTC
Pharmacokinetic data
Bioavailability40–45% (oral) [1]
Protein binding15–20% [1]
Elimination half-life2.5–3.5 hours [1]
ExcretionRenal (25-30% unchanged [Oral]) [1]

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The primary clinically important pharmacologic activity of famotidine is inhibition of gastric secretion. Both the acid concentration and volume of gastric secretion are suppressed by famotidine, while changes in pepsin secretion are proportional to volume output.Gastric emptying and exocrine pancreatic function were not affected by famotidine tablets.

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Minimally clinically significant. Minimize risk; assess risk and consider an alternative drug, take steps to circumvent the interaction risk and/or institute a monitoring plan.Lockey RF, Benedict LM, Turkeltaub PC, Bukantz SC.

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These differences were statistically significant.The incidence of DU healing with famotidine was greater than with placebo at each time point based on proportion of endoscopically confirmed healed DUs. Trials have not assessed the safety of famotidine in uncomplicated active DU for periods of more than 8 weeks.

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Famotidine is eliminated by renal 65 to 70% and metabolic 30 to 35% routes.The binding of famotidine in plasma proteins is relatively small 15-20% The plasma half-life is about 3 hours both after oral single doses and during repeated administration 5 days Up to 30-35% of famotidine is metabolised in the liver to famotidine 20mg vs ranitidine 150 mg inactive sulfoxide metabolite.

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The primary clinically important pharmacologic activity of famotidine is inhibition of gastric secretion. oth the acid concentration and volume of gastric secretion are suppressed by famotidine, while changes in pepsin secretion are proportional to volume p. divided b.

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Duration of inhibition of secretion by doses of 20 and 40 mg was 10 famotidine 20 mg vs ranitidine 150 mg 12 hours. Single evening oral doses of 20 and 40 mg inhibited basal and nocturnal acid secretion in all subjects; mean nocturnal gastric acid secretion was inhibited by 86% and 94% respectively, for a period of at least 10 hours.Data sources include IBM Watson Micromedex updated 10 Aug 2020 Cerner Multum updated 3 Aug 2020 Wolters Kluwer updated 10 Aug 2020 andothers.

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